Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis
This Special Issue of the International Journal of Molecular Sciences (IJMS), entitled “Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis”, collected 15 original research papers (5 reviews and 10 articles) written by a panel of experts from different countries who highlight...
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| Format: | Online |
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| Jezik: | angleščina |
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MDPI - Multidisciplinary Digital Publishing Institute
2024
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| Online dostop: | ONIX_20240514_9783725807024_413 |
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| _version_ | 1863747617025949696 |
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| collection | Directory of Open Access Books |
| description | This Special Issue of the International Journal of Molecular Sciences (IJMS), entitled “Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis”, collected 15 original research papers (5 reviews and 10 articles) written by a panel of experts from different countries who highlight recent advances in the EMT process.Navigating the complex field of EMT, this Special Issue introduces the current understanding of the underlying mechanisms of EMT in the evolution and progression of fibrogenesis and discusses potential strategies for attenuating EMT to prevent and/or inhibit fibrosis.Overall, the 15 scientific articles in this Special Issue of the International Journal of Molecular Sciences provide valuable insights into the complex mechanisms governing the EMT process linked to fibrosis and have highlighted the potential of novel therapeutic strategies. In the last few years, the field of EMT has shown considerable promise, and there is still much to be learned. As our understanding continues to grow, we hope that this Special Issue serves as a catalyst for further research and innovation in this developing field. |
| format | Online |
| id | doab-20.500.12854ir-137817 |
| institution | Directory of Open Access Books |
| language | eng |
| publishDate | 2024 |
| publishDateRange | 2024 |
| publishDateSort | 2024 |
| publisher | MDPI - Multidisciplinary Digital Publishing Institute |
| publisherStr | MDPI - Multidisciplinary Digital Publishing Institute |
| record_format | ojs |
| spelling | doab-20.500.12854ir-1378172024-05-14T14:34:38Z Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis Lisi, Sabrina Sisto, Margherita defense peptides immune peptides pulmonary fibrosis hypersensitivity pneumonitis extrinsic allergic alveolitis autoimmune thyroid diseases Graves’ disease Hashimoto’s Thyroiditis TGF-β EMT primary cilia mesenchymal stem cells mechanotransduction collagen oxidation YAP/TAZ focal adhesion proliferative vitreoretinal diseases (PVDs) experimental models epithelial-mesenchymal transition (EMT) preeclampsia placenta decidua MET 15-PGDH PGT signal transducer and activator of transcription 3 (STAT3) glaucoma filtration surgery fibrosis suppressor of cytokine signaling 3 (SOCS3) epithelial-to-mesenchymal transition chronic airway diseases remodelling collagen 1a1 bleomycin lansoprazole Mcp1 Mcp3 Opn idiopathic pulmonary fibrosis surfactant related gene mutation telomere related gene mutation SFTPC SFTPA2 RTEL1 TERT lung tissue RNA expression corylin anti-inflammation liver fibrosis hepatic stellate cell growth arrest-specific gene 6/AXL signaling pathway inflammation pulmonary malignancy natural products aggressiveness tumor stem cells mechanism of action fibroblast bioinformatics heterogeneity predictive model asthma airway epithelial to mesenchymal transition Cl− channel cell volume epigenetic DNA methylation histone modification ncRNA STAT4 STAT1 cytokine regulation interferons EGF n/a This Special Issue of the International Journal of Molecular Sciences (IJMS), entitled “Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis”, collected 15 original research papers (5 reviews and 10 articles) written by a panel of experts from different countries who highlight recent advances in the EMT process.Navigating the complex field of EMT, this Special Issue introduces the current understanding of the underlying mechanisms of EMT in the evolution and progression of fibrogenesis and discusses potential strategies for attenuating EMT to prevent and/or inhibit fibrosis.Overall, the 15 scientific articles in this Special Issue of the International Journal of Molecular Sciences provide valuable insights into the complex mechanisms governing the EMT process linked to fibrosis and have highlighted the potential of novel therapeutic strategies. In the last few years, the field of EMT has shown considerable promise, and there is still much to be learned. As our understanding continues to grow, we hope that this Special Issue serves as a catalyst for further research and innovation in this developing field. 2024-05-14T14:34:34Z 2024-05-14T14:34:34Z 2024 book ONIX_20240514_9783725807024_413 9783725807024 9783725807017 https://directory.doabooks.org/handle/20.500.12854/137817 eng application/octet-stream Attribution-NonCommercial-NoDerivatives 4.0 International https://mdpi.com/books/pdfview/book/9052 https://mdpi.com/books/pdfview/book/9052 MDPI - Multidisciplinary Digital Publishing Institute 10.3390/books978-3-7258-0701-7 10.3390/books978-3-7258-0701-7 46cabcaa-dd94-4bfe-87b4-55023c1b36d0 9783725807024 9783725807017 290 open access |
| spellingShingle | defense peptides immune peptides pulmonary fibrosis hypersensitivity pneumonitis extrinsic allergic alveolitis autoimmune thyroid diseases Graves’ disease Hashimoto’s Thyroiditis TGF-β EMT primary cilia mesenchymal stem cells mechanotransduction collagen oxidation YAP/TAZ focal adhesion proliferative vitreoretinal diseases (PVDs) experimental models epithelial-mesenchymal transition (EMT) preeclampsia placenta decidua MET 15-PGDH PGT signal transducer and activator of transcription 3 (STAT3) glaucoma filtration surgery fibrosis suppressor of cytokine signaling 3 (SOCS3) epithelial-to-mesenchymal transition chronic airway diseases remodelling collagen 1a1 bleomycin lansoprazole Mcp1 Mcp3 Opn idiopathic pulmonary fibrosis surfactant related gene mutation telomere related gene mutation SFTPC SFTPA2 RTEL1 TERT lung tissue RNA expression corylin anti-inflammation liver fibrosis hepatic stellate cell growth arrest-specific gene 6/AXL signaling pathway inflammation pulmonary malignancy natural products aggressiveness tumor stem cells mechanism of action fibroblast bioinformatics heterogeneity predictive model asthma airway epithelial to mesenchymal transition Cl− channel cell volume epigenetic DNA methylation histone modification ncRNA STAT4 STAT1 cytokine regulation interferons EGF n/a Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis |
| title | Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis |
| title_full | Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis |
| title_fullStr | Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis |
| title_full_unstemmed | Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis |
| title_short | Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis |
| title_sort | molecular mechanism in epithelial mesenchymal transition emt and fibrosis |
| topic | defense peptides immune peptides pulmonary fibrosis hypersensitivity pneumonitis extrinsic allergic alveolitis autoimmune thyroid diseases Graves’ disease Hashimoto’s Thyroiditis TGF-β EMT primary cilia mesenchymal stem cells mechanotransduction collagen oxidation YAP/TAZ focal adhesion proliferative vitreoretinal diseases (PVDs) experimental models epithelial-mesenchymal transition (EMT) preeclampsia placenta decidua MET 15-PGDH PGT signal transducer and activator of transcription 3 (STAT3) glaucoma filtration surgery fibrosis suppressor of cytokine signaling 3 (SOCS3) epithelial-to-mesenchymal transition chronic airway diseases remodelling collagen 1a1 bleomycin lansoprazole Mcp1 Mcp3 Opn idiopathic pulmonary fibrosis surfactant related gene mutation telomere related gene mutation SFTPC SFTPA2 RTEL1 TERT lung tissue RNA expression corylin anti-inflammation liver fibrosis hepatic stellate cell growth arrest-specific gene 6/AXL signaling pathway inflammation pulmonary malignancy natural products aggressiveness tumor stem cells mechanism of action fibroblast bioinformatics heterogeneity predictive model asthma airway epithelial to mesenchymal transition Cl− channel cell volume epigenetic DNA methylation histone modification ncRNA STAT4 STAT1 cytokine regulation interferons EGF n/a |
| topic_facet | defense peptides immune peptides pulmonary fibrosis hypersensitivity pneumonitis extrinsic allergic alveolitis autoimmune thyroid diseases Graves’ disease Hashimoto’s Thyroiditis TGF-β EMT primary cilia mesenchymal stem cells mechanotransduction collagen oxidation YAP/TAZ focal adhesion proliferative vitreoretinal diseases (PVDs) experimental models epithelial-mesenchymal transition (EMT) preeclampsia placenta decidua MET 15-PGDH PGT signal transducer and activator of transcription 3 (STAT3) glaucoma filtration surgery fibrosis suppressor of cytokine signaling 3 (SOCS3) epithelial-to-mesenchymal transition chronic airway diseases remodelling collagen 1a1 bleomycin lansoprazole Mcp1 Mcp3 Opn idiopathic pulmonary fibrosis surfactant related gene mutation telomere related gene mutation SFTPC SFTPA2 RTEL1 TERT lung tissue RNA expression corylin anti-inflammation liver fibrosis hepatic stellate cell growth arrest-specific gene 6/AXL signaling pathway inflammation pulmonary malignancy natural products aggressiveness tumor stem cells mechanism of action fibroblast bioinformatics heterogeneity predictive model asthma airway epithelial to mesenchymal transition Cl− channel cell volume epigenetic DNA methylation histone modification ncRNA STAT4 STAT1 cytokine regulation interferons EGF n/a |
| url | ONIX_20240514_9783725807024_413 |